Fig. 1

Magnetic resonance axial SE T1WI (Fig. a, c) and DWI (Fig. b, d) of a patient with recurrent multiple acute cerebral infarcts. The patient was admitted to the hospital for the first time, and an MRI showed multiple acute infarction lesions in the left radiocaudate region and the left parietal lobe, with long T1 signals (Fig. a) and high DWI signals indicative of restricted diffusion (Fig. b). The patient developed clinical symptoms six months after discharge from rehabilitation, an MRI showed multiple acute infarction lesions in the left radiocaudate region with long T1 signals and multiple areas of infarction were also seen in the left radiocaudate region (Fig. c); diffusion-limited DWI with a high signal (Fig. d)

Fig. 2

Magnetic resonance axial SE T1WI (Fig. a, c) and DWI (Fig. b, d) of a patient with recurrent single lesion of acute stroke.The patient was admitted to the hospital for the first time, and an MRI showed a single lumen infarction in the left external capsule with a long T1 signal (Fig. a) and diffusion-limited DWI with a high signal (Fig. b). The patient developed clinical symptoms four months after discharge from rehabilitation, an MRI showed a single lumen infarction in the right parietal lobe with a long T1 signal (Fig. c) and diffusion-limited DWI with a high signal (Fig. d)

Fig. 3

Magnetic resonance axial SE T1WI image and a schematic diagram outlining the subcutaneous fat area

Fig. 4

Magnetic resonance axial SE T1WI (a), DWI and the schematic diagram of the maximum lesion area measurement at the largest layer (b). In (a), MRI showed an acute infarction lesion in the head of the left caudate nucleus, presenting as long T1 signals; In (b), DWI showed restricted diffusion with high signal intensity.

Table 1

Comparison of general data between the recurrent and non-recurrent stroke groups

Table 2

Comparison of MRI data between the recurrent and non-recurrent groups of cerebral infarction